Oral and Maxillofacial Surgery Lectures

Complications Associated with Dentoalveolar Surgery

Complications Associated with Dentoalveolar Surgery

Complications Associated with Dentoalveolar Surgery

Dentoalveolar surgery, while routine, carries the risk of several postoperative complications. A thorough understanding of their etiology, risk factors, prevention, clinical features, and management is crucial for both dental students and practicing clinicians.

Dental MCQs – Complications in Dentoalveolar Surgery

1. Postoperative Bleeding

Etiology & Prevalence
✦ Bleeding is a common side effect of dentoalveolar procedures.
✦ In healthy patients, it is usually minimal and self-limiting due to clot formation.

Types:

  • Oozing: minor, resolves within 36–72 hours, controlled by pressure.
  • Active bleeding: profuse, mouth fills rapidly with blood once gauze is removed.

Risk Factors & Prevention
✔ Careful medical history is critical:

  • Systemic disorders: hemophilia, von Willebrand disease.
  • Medications: aspirin, clopidogrel, warfarin, heparin, newer anticoagulants (rivaroxaban, apixaban, dabigatran).
  • Family history of bleeding.
  • Female patients with menorrhagia.

✔ Notes on anticoagulants:

  • Aspirin/NSAIDs/clopidogrel → usually safe to continue.
  • Warfarin: safe if INR < 2.5 (multiple extractions) or < 3.0 (1–3 simple extractions).
  • Consider staged visits.
  • New anticoagulants: safer, fixed dosing, but costly.

Treatment

  • Intraoperative: gentle tissue handling, vasoconstrictors, primary closure, topical hemostatics.
  • Postoperative: gauze pressure, remove “liver clot,” vasoconstrictors, hemostatic packs + sutures.
  • Severe cases: arterial ligation, electrocautery, or embolization.

2. Postoperative Pain

Etiology & Prevalence

  • Onset: 6–12 hours post-op (after LA wears off).
  • Peaks: 24–48 hours.

Prevention & Management
✔ Minimize surgical trauma and flap tension.
✔ Pre-op NSAIDs (salicylates, COX-2 inhibitors).
✔ Post-op:

  • NSAIDs = first line.
  • Opioids + acetaminophen (hydrocodone, oxycodone, tramadol) for severe pain.
  • Long-acting anesthetics (bupivacaine + epinephrine) to delay onset.

3. Postoperative Swelling

Etiology & Prevalence

  • Common and expected.
  • Onset: 12–24 hrs → peaks: 48–72 hrs → subsides by day 4 → resolves within 1 week.

Prevention & Management
✔ Inform patients swelling is normal.
✔ Ice packs in first 24 hrs.
✔ Head elevation during sleep.
✔ Corticosteroids (for extensive surgery, e.g., 3rd molars).

4. Surgical Site Infection

Etiology & Prevalence

  • Oral cavity contains mixed flora → risk of infection.
  • More common with mandibular 3rd molars.

Risk Factors

  • Older age, smoking, oral contraceptives, poor hygiene, pre-existing infection, surgical trauma.

Prevention

  • Atraumatic technique, thorough debridement, irrigation, removal of necrotic tissue.

Treatment

  • Symptoms: persistent pain, swelling, pus, trismus, foul taste, ± fever.
  • Early cellulitis: broad-spectrum antibiotics.
  • Abscess: incision & drainage + C&S.
  • Severe spread: risk of airway compromise → emergency management.

📊 Summary Table: Common Postoperative Complications

ComplicationOnsetPeak/DurationMain PreventionKey Management
BleedingImmediateHours post-opRisk assessment, hemostatic measuresGauze, vasoconstrictors, sutures, arterial control
Pain6–12 hrs24–48 hrsNSAIDs, gentle techniqueNSAIDs, opioids + APAP, long-acting LA
Swelling12–24 hrs48–72 hrsIce, elevation, steroidsReassurance, symptomatic care
InfectionVariable (days)ProgressiveDebridement, irrigation, risk assessmentAntibiotics, I&D, airway protection if severe

5. Alveolar Osteitis (Dry Socket)

Prevalence & Etiology
✦ One of the most frequent complications after extractions, esp. impacted teeth.
✦ Incidence up to 30%.
✦ Caused by dislodgment or failure of clot formation (not infection).

Clinical Features

  • Severe throbbing pain (3–5 days post-op).
  • Halitosis.
  • Trismus (from pain).
  • Empty socket with exposed bone, erythematous margins, food debris.
  • No fever or swelling.

Prevention

  • Risk factors: age, smoking, poor hygiene, oral contraceptives, infection, mandibular extractions, inexperience.
  • Measures: socket irrigation, medicaments (e.g., Gelfoam®, PRP, medicated rinses).

Treatment
✔ Self-limiting – focus on pain control.

  • Warm saline irrigation.
  • Medicated dressings (eugenol packs, changed every 24 hrs).
  • Analgesics.

6. Fractures

Prevalence & Etiology
✦ Rare but serious; due to excessive extraction force.
✦ May affect alveolar bone or mandible → malocclusion, malunion, paresthesia, infection.

Prevention

  • Higher risk in elderly (low bone density).
  • Atrophic mandibles or with large lesions = vulnerable.

Treatment

  • Clinical recognition: mobile segments, malocclusion, disproportionate pain/swelling.
  • Imaging: periapical, panoramic, CT.
  • Mild: dietary modification, immobilization.
  • Severe: reduction and fixation.

7. Root Fractures

Prevalence & Etiology
✦ Common with multirooted posterior teeth.
✦ Caused by excessive force, poor root separation, root anatomy variations.

Prevention

  • Correct technique, sectioning when needed.
  • Recognize risky roots (curved, dilacerated, thin).

Treatment

  • Inspect extracted tooth for completeness.
  • Small, asymptomatic, <3 mm root tips without infection → may be left (coronectomy).
  • If pathology present → careful removal with root tip picks, avoiding apical pressure.

8. Root or Tooth Displacement

Prevalence & Etiology
✦ Rare but distressing.
✦ Maxillary → fragments into sinus or infratemporal fossa.
✦ Mandibular → fragments into submandibular space or IAN canal.

Prevention

  • Proper surgical technique (periosteal elevator as barrier).
  • Radiographs to assess sinus/root proximity.

Treatment

  • Maxillary sinus: localize via imaging, retrieval by sinus pressure, suction, gauze packing, or surgical exploration. Post-op antibiotics + sinus precautions.
  • Mandibular: immediate imaging. Gentle removal if possible; otherwise, delay until fibrosis stabilizes fragment. IAN canal fragments may be left if asymptomatic.

9. Oroantral Communication (OAC)

Prevalence & Etiology
✦ Common after maxillary posterior extractions.
✦ <1% persist as fistulas.
✦ Caused by close anatomical root–sinus relationship.

Prevention

  • Radiographic evaluation pre-op.
  • Gentle curettage, avoid excessive apical pressure.
  • Inform patient of risk if high.

Diagnosis

  • Valsalva maneuver → air bubbles in socket.

Treatment

  • Small defects: usually heal spontaneously.
  • Supportive meds: antibiotics (amoxicillin, clindamycin), sinus precautions, nasal decongestants.
  • Persistent OAC/fistula: surgical closure with layered repair + sinus management.

Dental MCQs – Complications in Dentoalveolar Surgery

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